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MRI THORACIC SPINE

Hello got the Thoracic Mri... NOT good.. Here i GO LOL... there is a mild accentuation of the usual thoracic kyphosis with no spondylolisthesis or scoliosos. THE thoracic vertebral bodies are normal in morphology with no acute or chronic fracture. There are two small hemangiomass within the inferior endplate of T3 throughthe superior endplate of L1.
there is a mild interrvertebral disc desiccation diffusely through the thoracic spine. 
Additional changes of the thoracic spine ona level by level basis are as follows.

T1-T2 throughT3-T4: No significant abnormality or stenosis.

T4-T5 small and shallow central disc protrusion which slightly impinges upon the ventral thecal sac without significant spinal canal stenosis or neural foramen stenosis.

T5-T6 minimal/mild bilateral degenerative facet arthrosis.

T6-T7 subtle small and shallow right paracentral disc protrusion and minimal/mild bilateral degenerative facet arthrosis with nospinal canal or neural foramen narrowing.

T7-T8.Subtle small and shallow,thin left paracentral extrusion extending slightly cranially from the level of the disc andmidly impinging upon the ventral thecal sac without significant spinal canal stenosis. There is also minimal degenerative facet changes on the right.

T8-T9 Subtle posterior annular bulging and minimal/mild bilateral degenerative facet arthrosis without spinal canal or neural foramen stenosis.

T9-T10 Minimal/mild bilateral degenerative facet artheosis without spinal canal or neural foramen stenosis.

If you want to know the rest hit me up lol

M.R.I 3-22-2018

Findings

There is moderate asymmetric left l5-S1 foraminal stenosis, a combination of uncovered disc,disc bulging and facet hypertrophic signal alteration.

 

There is a concentric disc bulge at L4-5, asymmetric signal alteration left facet hypertrophy produces mild left greater than right foraminal stenosis.

Also Multisegment Schmorl's nodes are present incidentally.

A little about me

Hi here is a little about me you may not know. I have been married twice and divorced.

I am 54 with no children but love kids. i havent dated anyone since 2009. i have Grave's Disease if you don't know what that is it has to do with not having a thyroid i have it in my eyes but you can get it in any muscle. i am very easy to talk too but do not want drama period.i hurt my low back doing a job i loved working in a nursing home back in 1990. My ex was very abusive and left me in indiana where i worked 2 full time jobs to get back on my feet then was declared disable in 2003. i do have a lot of pain i do not do pain pills because i am allergic to most. 

 I am from Texas but live in louisiana renting a room from a friend.. i have had major foot surgery still having a lot of problems with that foot now my doctor thinks i might be starting to have RA.. after i see the RA doctor might be having more foot surgery to shorten a bone and fix around my ankle it is bone ontop of bone.

Will accept any and all friends

Im sorry if i am not a good writter lol


What Is Grave's Disease

Graves’ disease is named for the doctor who first described it in Ireland—Robert J. Graves. He noticed it in a patient in 1835. The disease is also referred to as Basedow’s disease—named after a German, Karl Adolph van Basedow, who described the disease in 1840. He didn’t know that Graves had described the same disease just a few years earlier. The term Basedow’s disease is more commonly used in continental Europe; in the United States, it’s called Graves’ disease.

 
Graves’ disease is a type of autoimmune problem that causes thethyroid gland to produce too much thyroid hormone, which is calledhyperthyroidism. Graves’ disease is often the underlying cause of hyperthyroidism.
 
Thyroid GlandAutoimmune problems—of which there are many different types—develop when your immune system causes disease by attacking healthy tissues. Researchers do not completely understand what causes autoimmunity, although there seems to be a genetic connection, as cases of Graves’ disease tend to run in families. For unknown reasons, like many autoimmune diseases, Graves’ is also more likely to affect women than men.
 
In Graves’ disease, your immune system creates antibodies that cause the thyroid to grow and make more thyroid hormone than your body needs. These antibodies are called thyroid-stimulating immunoglobulins (TSIs). The TSIs bind to thyroid cell receptors, which are normally “docking stations” for thyroid-stimulating hormone (TSH—the hormone responsible for telling the thyroid to produce hormones). The TSIs then trick the thyroid into growing and producing too much thyroid hormone, leading to hyperthyroidism.
 
Symptoms
The early symptoms of Graves’ disease may be confused with other conditions and make diagnosis a challenge. Some of the more common symptoms include:
  • Weight loss—despite increased appetite
  • Anxiety, restlessness, tremors, irritability, difficulty sleeping (insomnia)
  • Heat intolerance, sweating
  • Chest pain, palpitations
  • Shortness of breath, difficulty breathing
  • Increased stool frequency (with or without diarrhea)
  • Irregular menstrual periods
  • Muscle weakness
  • Difficulty controlling diabetes
  • Goiter
  • Prominent, bulging eyes
  • Vision problems (such as double vision)
 
Physical Signs and Symptoms
If Graves’ disease goes untreated, physical signs and symptoms may develop.
  • Goiter: goiter is an enlarged thyroid gland. A goiter related to Graves’ disease is a diffuse thyrotoxic goiter. As the thyroid gets bigger, the patient’s neck may begin to look full or swollen. Sometimes a goiter makes swallowing difficult, causes coughing, and may disrupt sleep.

 

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